By A Henry (Arnold Henry) Savage-Landor
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Extra resources for Across Coveted Lands; Or, a Journey from Flushing (Holland) to Calcutta, Overland; Volume 1
1985). , 1990). , 1985). Second, known teratogens produce a more or less specific pattern of defects; isolated anomalies are not the rule. Thus, one would not expect a teratogenic agent to induce cleft palate in one case, limb defects in another, or closure abnormalities in still another. A teratogen might produce a single specific or a whole pattern or syndrome of defects, but these would have some degree of uniformity from case to case, if chemically induced. , tail defects) should not give comfort in interpretation.
IUGR is not uncommon in infants with severe and multiple malformations (Warkany, 1971). In addition, overwhelming congenital infections, neonatal deaths, and long-term neurological and intellectual deficits have been recorded more frequently than among infants with normal growth (Miller, 1981). , 1979). In humans, IUGR is a different entity than small-for-gestational-age (SGA) newborns, and the two should be distinguished from each other (Wilcox, 1983; Goodlin, 1990). C. Embryolethality Death of offspring is another class of developmental toxicity.
1970). , 1981). Limb, jaw and gonadal defects occur most frequently in sheep (Dennis, 1974). Various types of defects are observed in humans. Approximations of population incidences for the most common of these are listed in Table 1-8. Ethnic, racial, and geographic factors influence the incidence figures appreciably (Warkany, 1971). For example, discounting polydactyly, common to black infants, one usually finds incidence figures lower among blacks than whites. Anencephaly occurs frequently in Ireland and Scotland, whereas it is rare in other localities.